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I have suffered from dry eye since taking roaccutane four and a half years ago. It has made my life a struggle, compounded by the fact that neither I nor ophthalmologists had a really clear idea of what had actually happened to me.
I was struck off by moorfields because my eyes looked 'fine', and even the private specialist dry eye centre in London thought my meibomian glands looked fine due to the meibography which revealed them to be perfectly intact. Erroneously, they (and, as a result, I), thought I had aqueous deficiency for years - mainly because a lot of experts kept saying my glands 'looked' fine.
Finally, after years of scouring the internet for real answers, I found an academic report that explains in detail what has happened to us, and what our treatment options are. Our type of MGD is different to any other. I had to pay $50 to access the journal which this report is in, and in so doing I learnt more than I ever did from the £10,000+ I have spent on private appointments thus far. The article explains why specialists have found it impossible to correctly diagnose us.
A basic overview
Our MGD does not appear the same as any other kind of MGD; eg, we appear asymptomatic on meibographys, our lid margins are not red, glands are not often blocked, and it is possible to express meibum from the glands.
Isotretinoin has no effect on the lacrimal glands
Gland function reduced by up to 90% - stays permanent in approx 1% of cases.
I have uploaded the full document here.
It is essential if you suffer from roaccutane induced dry eye that you send this to your ophthalmologists in advance of appointments so you are not misdiagnosed, and they understand what they are dealing with and what they should be looking for.
I have uploaded the document here.
https://www.scribd.com/document/3192...ibomian-Glands
I was struck off by moorfields because my eyes looked 'fine', and even the private specialist dry eye centre in London thought my meibomian glands looked fine due to the meibography which revealed them to be perfectly intact. Erroneously, they (and, as a result, I), thought I had aqueous deficiency for years - mainly because a lot of experts kept saying my glands 'looked' fine.
Finally, after years of scouring the internet for real answers, I found an academic report that explains in detail what has happened to us, and what our treatment options are. Our type of MGD is different to any other. I had to pay $50 to access the journal which this report is in, and in so doing I learnt more than I ever did from the £10,000+ I have spent on private appointments thus far. The article explains why specialists have found it impossible to correctly diagnose us.
A basic overview
Our MGD does not appear the same as any other kind of MGD; eg, we appear asymptomatic on meibographys, our lid margins are not red, glands are not often blocked, and it is possible to express meibum from the glands.
Isotretinoin has no effect on the lacrimal glands
Gland function reduced by up to 90% - stays permanent in approx 1% of cases.
I have uploaded the full document here.
It is essential if you suffer from roaccutane induced dry eye that you send this to your ophthalmologists in advance of appointments so you are not misdiagnosed, and they understand what they are dealing with and what they should be looking for.
I have uploaded the document here.
https://www.scribd.com/document/3192...ibomian-Glands
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