At the risk of asking the obvious: would you guys agree that the feeling we often describe as "that menthol sensation," or "like a raw wound" is, in effect, the result of evaporative dry eye, i.e. surface irritation?
So many of us, especially post-LASIK, get this MGD diagnosis. We have relatively good aqueous production (Schirmers) but cruddy lipids (TBUT) - the water's coming out, but there's nothing holding it to the eye. We get too much surface exposure, the veins pop out and - presto! - "that menthol sensation."
Ergo: the approach to attack this, and God willing reverse some of it, is to boost the lipids, every way possible - fish oil, lid massage/gland expression etc.
(Or, do we think that "sensation" perhaps originates more from the damaged nerves at the site of the interface? This is a source of much confusion for me!)
Thanks,
Rob
So many of us, especially post-LASIK, get this MGD diagnosis. We have relatively good aqueous production (Schirmers) but cruddy lipids (TBUT) - the water's coming out, but there's nothing holding it to the eye. We get too much surface exposure, the veins pop out and - presto! - "that menthol sensation."
Ergo: the approach to attack this, and God willing reverse some of it, is to boost the lipids, every way possible - fish oil, lid massage/gland expression etc.
(Or, do we think that "sensation" perhaps originates more from the damaged nerves at the site of the interface? This is a source of much confusion for me!)
Thanks,
Rob
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